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Its core mechanism involves actively depleting glucose within the tumor microenvironment (TME), thereby disrupting the dysfunctional Warburg effect that tumor cells rely upon while simultaneously remodeling the immune microenvironment

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This is different from exposure to carcinogenic and genotoxic mycotoxins which can have a long-term negative impact on human health

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Furthermore, the over-expression of the ceruloplasmin lncRNA (NRCP) in ovarian and breast cancer cells, along with the LINK-A lncRNA in triple negative breast cancer, promotes glucose uptake, favoring lactate production and consequently, enhancing tumor progression (Rupaimoole et al., 2015