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Mitochondria, NADPH oxidases and xanthine oxidases have all been identified as potential contributors to ROS production, yet the exact redox mechanisms underlying exercise-induced oxidative stress remain elusive

En humanos, los datos son ms limitados: A dosis bajas (equipotentes sobre GH): la selectividad parece mantenerse A dosis altas o en uso prolongado : no hay ensayos humanos robustos que confirmen la selectividad a escalas operativamente relevantes Esta es una salvedad metodolgica importante