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The data also show that cellular thiol concentrations were augmented in the presence of GSH and NAC and these compounds were cytoprotective against HgCl 2 and this is due to up regulating of GSH synthesis

Souhaitons que cette explication rduise en partie la confusion, plutt que den crer davantage

Collection of blood biochemical data and animal tissues Phenotypic data of 20 blood parameters (i.e., blood oxygen saturation, glucose, triglycerides, alanine transaminase, aspartate aminotransferase, total bilirubin, alkaline phosphatase, lactate dehydrogenase, uric acid, blood urea nitrogen, creatinine, cardiac enzymes, calcium, superoxide dismutase, glutathione peroxidase, catalase, nitric oxide, malondialdehyde, erythropoietin and nitric oxide synthase) and samples from 19 whole-body tissues [i.e., heart, liver, spleen, lung, kidney, rumen, abomasum, duodenum, ileum, jejunum, colon, cerebrum, cerebellum, hypothalamus, pituitary, artery, muscle, adipose and blood (leukocyte)] were collected from all the animals in the above three scenarios

Congestive liver disease refers to a spectrum of liver abnormalities attributed to the passive congestion secondary to right-sided heart failure or any cause that increases the central venous pressure, including volume overload, severe pulmonary hypertension or valvulopathies.28 Elevated serum GGT or ALP are characteristic biochemical abnormalities in CKD patients with left ventricular diastolic dysfunction, especially if they are associated with pulmonary congestion,29 which is also a recognised risk factor for mortality in these patients.30 Elevated GGT can also reflect microsomal enzyme induction.31 In addition to alcohol intake, many drugs can induce liver enzymes

The implication of this structural observation will be discussed later