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In humans and rodents, the soluble GSTs are collectively expressed in relatively large amounts, and may comprise as much as 4% of total soluble protein in the liver [58]

Ferroptosis mediates inflammation, oxidative stress, and lipid oxidative damage processes, and also participates in the occurrence and pathological progression of inflammatory joint diseases including RA

848 These suggest that NCOA4 may contribute to intestinal I/R injury in mice by inducing ferroptosis through mediating ferritinophagy

Regenerating this mutation with CRISPR demonstrated that it was the causative mutation, as described in the text

doi: 10.1177/1535370214533880 100 NamKWJeKHLeeJHHanHJLeeHJKangSKet al