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It is tempting to speculate that an attenuated anti-oxidant response with elevated ROS in COPD [21, 22, 28, 36] may lead to an increased oxidant burden, possibly contributing to the observed mitochondrial defects in bronchial epithelial cells from COPD patients [6, 32, 37]

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Since so few larvae reached this instar in the sediment treatment groups, a considerable finding in itself, teratogenicity was assessed in the control and water treatment groups only

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The ash and paper part of the cigarettes gets composted