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Description

This case report outlines the evaluation of a patient in a Metabolic Bone Disease Clinic at The Aga Khan University Hospital, Karachi, Pakistan

Wan, H., Adams, R

This mutation results in protein accumulation within the endoplasmic reticulum (ER) of hepatocytes and other AAT-producing cells, which leads to ER stress or an aberrant inflammatory response

This environmental persistence is exacerbated by its slow decomposition rate, with a half-life estimated between 95 and 350 days in the water column 5