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treated with N-acetylcysteine (NAC)

The role of tumour necrosis factor in neuroinflammation associated with parkinsons disease and targeted therapies

Genetic predispositions in glutathione metabolism (GST polymorphisms, SOD variants) can inform whether enhanced antioxidant support aligns with individual physiologythough genetics alone do not predict clinical response to supplementation

Prolonged exposure to stress further elevates the activity of monoamine oxidase-A (MAO-A) while concurrently diminishing monoamine biosynthesis, thereby exacerbating depressive-like manifestations (26)