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Lp(a) levels less than 10 mg/dL (18 nmol/L) had the lowest risk.[ref] 28% increased relative risk of major cardiac event with Lp(a) 10 to 49 mg/dL (18 105 nmol/L) 44% increased relative risk of major cardiac event with Lp(a) between 50 to 99 mg/dL (105213 nmol/L) 114% increased relative risk (a bit more than double the risk) of major cardiac event with Lp(a) 100 mg/dL (214 nmol/L) In women specifically: A 2026 study involving almost 30,000 women showed that the risk of major cardiac events increased almost linearly starting at Lp(a) levels over 30 mg/dL

Fecal excretion of total carnitine was less than 1% of the administered dose.10 After attainment of steady state following 4 days of oral administration of CARNITOR Tablets (1980 mg q12h) or Oral Solution (2000 mg q12h) to 15 healthy male volunteers, the mean urinary excretion of levocarnitine during a single dosing interval (12h) was about 9% of the orally administered dose (uncorrected for endogenous urinary excretion)

Moreover, elevated systemic concentrations of TMAO enhanced thrombosis potential in vivo in animal models (4), and in recent clinical intervention studies, elevation in plasma TMAO induced with dietary choline supplementation altered platelet hyperresponsiveness in subjects (omnivores and vegans/vegetarians alike), as monitored by platelet aggregometry (17)

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