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Additionally, when fatty acid oxidation was blocked by etomoxir, MST6-I cells exposed to secretomes from white or TWAT(MST6-I) adipocytes showed a compensatory increase in glycolytic flux (Figure 4B, right panel), demonstrating a preferential reliance on fatty acid oxidation under substrate exposure

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3C, G), a higher level of 4-HNE staining (Fig

Cancer 106 , 14671468 (2012)

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