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Mitochondrial homeostasis is considered the key parameter for objective measurements of the patients health status (optimal versus suboptimal one), therapy success monitoring, coaching of adapted physical activities, and cost-effective pre- and rehabilitation performance

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Surprisingly, mice with specific genetic deletion of Gpx1 appear phenotypically normal and with normal life span (Ho et al., 1997), suggesting that there are alternative compensatory mechanisms for H 2 O 2 scavenging in Gpx1 deficiency

Rescue of cells from apoptosis by inhibition of active GSH extrusion