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In addition to the aforementioned mechanisms, other mechanisms like protease/antiprotease imbalance, mucus hypersecretion, and cellular senescence also play significant roles in the pathogenesis and progression of COPD

Especially in those who are genetically predisposed, oxidative stress strengthens the relationship between environmental factors and the early stages of autoimmune condition

Prevention of radical metabolite production Scavenging of produced radicals Repair of cell damage Stopping chain reactions that produce secondary radicals Enhancement of endogenous antioxidant capacity is mediated through 5 different channels

Bone marrow stromal cells protect myeloma cells from bortezomib induced apoptosis by suppressing microRNA-15a expression

doi: 10.3390/antiox12020416 499 SabadashkaMHertsykDStrugaa-DanakPDudekAKanyukaOKucharskaAZet al