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Oxidative stress is a known factor in how the body manages airway sensitivity and inflammation

In addition, it disrupts oxidative phosphorylationmediated cuproptosis and metabolic reprogramming through mitochondrial proteolipid acylation, thereby impairing insulin and energy metabolism (58, 82, 83) and accelerating the progression of DM and its complications (57, 84)

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We also correlated dystonia severity with post-mortem measures of residual striatal dopamine and nigral neuron counts obtained a mean 53 15 days following MPTP, after resolution of dystonia but during stable parkinsonism