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Deoxycholic acid, for example, signals through TGR5, leading to PKA inhibition and has been linked to EGFR activation, ERK1/2 signaling, and occludin redistribution in Caco-2 cells (57, 58)

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Here we have elucidated the complex crosstalk between the TGF-- and HGF-dependent signaling pathways in gliobastoma using three patient-derived GIC models as well as human glioblastoma specimens

At later stages of the disease, low doses of the peptide also reduced the level of T cells, drivers of the immune response and inflammation, moving into the area

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