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1c,d) which catabolize 25(OH)VD 3 and active 1,25(OH) 2 D 3

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The excellent clinical manifestations of Tan IIA provide supportive evidence for targeting the GSH system in the treatment of heart diseases, which may be promising for transforming natural compounds from basic research to clinical applications

Unbound iron enters the cardiomyocytes through the L-type calcium channels ( The fact that cardiomyocyte iron overload is a storage problem and not an infiltrative process indicates that iron can be removed to reverse cardiac iron overload and associated damage ( Molecular mechanism of myocardial injury caused by cardiac iron overload in thalassemia Iron overload is a common feature in patients with -thalassemia, but the molecular mechanism of cardiac damage caused by iron overload in thalassemia is poorly understood

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