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A major influx of inflammatory cells is commonly regarded as the primary event during the pathogenesis of allergic asthma [45]

This mechanism is particularly relevant in autoimmune conditions where Treg dysfunction contributes to disease pathogenesis (Singer et al., 2024)

Human pituitary responsiveness to secretagogues is also affected by age, sex, body composition, and somatostatin tone, none of which are controlled for in typical compounding protocols

To further clarify these mechanisms, the Keap1/Nrf2/ARE signaling pathway was explored