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It has been demonstrated in vitro that amygdalin can effectively inhibit Ang II-induced cardiomyocyte hypertrophy, reduce inflammatory response, and exhibit anti-oxidant activity when treating H9C2 cells induced by Ang II at concentrations of 80, 160, and 320 M

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only a few negligible amounts of radio-labeled bovine GH are transported to the mitochondria compared with other subcellular compartments (Mutvei et al., 1989)

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