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Although the molecular role that reduced homocysteine plays in vascular disorders is unclear, experimental evidence indicates that the association between moderately elevated homocysteine levels and atherothrombotic disease is likely to be causal

( in vitro assays observing that acrolein increases neuronal excitation

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Another important gap is that most animal studies assess preventive or very early interventions, whereas patients usually present after organelle injury, inflammation, and systemic complications are already established