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Indeed, microglial activation leads to increased production of cytokines and activation of nitric oxide synthases (iNOS)

They do not show that BPC-157 binds directly to FAK or that the same pathway controls every effect in every cell type

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Alternatively, it was suggested that the discrepancies in the presence of ROS would be explained by differences in the timepoints studied, in a scenario where oxidative stress would be a secondary effect rather than a primary cause, occurring later in the progression of FRDA (discussed in Lupoli et al., 2018)