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These disruptions lead to water and sodium influx, potassium efflux, cell swelling, membrane potential changes, and eventual cell lysis, which then releases intracellular contents, eliciting an inflammatory phenotype through DAMP release and triggering immune responses (Murao et al., 2021)

Beyond personalization, these systems play a critical role in anticipating and counteracting drug resistance

Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB

Funding This work is supported by National Science Foundation, Division of Chemical, Bioengineering, Environmental and Transport Systems, Energy for Sustainability Program

coli strain BL21 (DE3) (Sigma, CMC0014) and purified as previously described 48