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The age-induced decline of aversive olfactory memories provides a powerful model system for scrutinizing molecular and cellular mechanisms provoking AMI in the Drosophila system.

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The flat bench showed strong middle and lower chest activation, while upper chest activity was highest at a moderate incline, not the steepest one tested

Treatment with etamicastat normalized the expression of these NOX isoforms but did not affect HO-1 expression in D 2 / mice (Fig