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Research progress in skin aging and immunity

However, recent genome-wide association studies (GWAS) have not consistently validated these links

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In recent decades, a broad range of studies has shown that the development of age-dependent neurodegeneration is due to a decrease in the antioxidant efficacy and an increase in oxidative damage

Figure 4 In parallel, an alternative ferroptosis defence pathway is mediated by ferroptosis suppressor protein 1 (FSP1), which operates independently of GPX4 by reducing coenzyme Q10 (CoQ10) to its active antioxidant form, ubiquinol