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Figure 7 diagrams how such interventions have been applied in different organs (heart, brain, kidney) and what outcomes have been observed or expected, bridging mechanism to clinical translation

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T h 1 cells promote lesion development and plaque instability through the production of proinflammatory IFN and TNF

We reasoned that the cyanoacrylamide moiety may be important for the degradation activity

found that inhibition of mTOR overactivation modulates autophagy and increases the expression of post-synaptic density markers (PSD-95) and presynaptic vesicle proteins (SYP) to ameliorate anesthesia/surgery-induced memory and cognitive dysfunction by constructing a mice model (Gao et al., 2021)