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Bioche 160, 257268

doi: 10.1007/s11154-022-09778-x 29 TangJShanSLiFYunP

Targeting these dysregulated pathways represents a fundamental approach for therapeutic interventions and disruption of the TME, offering the potential to improve GBM treatment outcomes (Fig

G.EilerS.MorasD.GangloffJ.et al (1997)

The observed PKR siRNA-dependent release of HSV-1 restriction resulted in 2226% of viral growth compared to the untreated controls (Supplementary Figs