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Since the host-derived damage-associated molecular patterns (DAMPs), like bile acids, can directly engage GSDME to trigger pyroptosis, we reasoned that there might have certain endogenous metabolites capable of inhibiting GSDME activation

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Chemisch gesehen sind diese beiden Stoffe auch sehr hnlich und werden im Krper ineinander umgewandelt

Unique anatomic and physiological challengessuch as lung tissue heterogeneity, air-tissue interfaces, and susceptibility to radiation-induced pneumonitisnecessitate tailored approaches