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Target cells in contact with wild-type attacker cells generally failed to proliferate and divide, whilst isolated target cells or target cells in contact with T6SS-deficient or Ssp4-lacking attackers proliferated indistinguishably from attacker cells (Fig

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Through inflammatory cell infiltration (neutrophils, T/B cells), cytokine network imbalance, and vascular wall structural destruction, it ultimately leads to vascular stenosis, occlusion, or rupture, resulting in tissue and organ ischemia or hemorrhage (210, 211)