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IL-1 promotes chondrocyte apoptosis and inhibits the synthesis of cartilage matrix components, whereas TNF- exacerbates joint inflammation and cartilage damage by stimulating the production of inflammatory mediators like nitric oxide and prostaglandin E2 [21, 22]

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Mitochondrial genes were removed from the analysis before integration

Increased funding accelerates innovation, clinical trials, and commercialization, creating a positive feedback loop that propels market growth and attracts further capital inflows