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This delay helps avoid the nutritional competition that can occur between a young woman and her developing foetus, allowing her more time to mature and potentially improving her nutritional status

10.1038/ejhg.2016.139 94 MoujalledD.StrasserA.LiddellJ

A specific oxidoreductase, previously known as apoptosis-inducing-factor mitochondrial-2 (AIFM2), capable of recycling reduced ubiquinol (Co-enzymeQ 10 H 2 ) from ubiquinone at the expense of NAD(P)H, has been presented as a potential ferroptosis inhibitor due to the fact that its overexpression complements the loss of GPx4 in PFA1 and human fibrosarcoma (66, 67)

Furthermore, increased E2 production can maintain follicular structure and performance, leading in greater follicle growth [14] (Fig

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