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Although aldosterone-induced oxidative or nitrative stress quickly stimulated antioxidant or detoxifying enzymes such SOD, thioredoxin (TRX), HO-1, or GCSc, this adaptive survival response appeared to be fleeting and overpowered by a chronic increased generation of ROS/RNS

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(2004) showed that abnormal neuronal and mitochondrial proteins induce swelling, vacuolization, and increased ROS levels intracellularly, and manifests as learning and memory deficits (Shin et al., 2004, p

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falciparum (PfTrxR, with an N-terminal 6-His tag was synthesized and inserted into pET15b (GenScript) and expressed in BL21 (DE3) cells and purified as described for human GR 20