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The share approached 15% by week 88 in the longest trial, while clinical underweight remained below 0.5%. The signal clustered in lower-baseline deep responders

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Paolicelli RC, Bolasco G, Pagani F, Maggi L, Scianni M, Panzanelli P, et al

-HB prevents the reduction of cytoplasmic potassium ion concentrations, thereby inhibiting NLRP3 activation, which would otherwise trigger caspase-1 activation and subsequent pro-inflammatory cytokine release in response to potassium depletion [36, 43]