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2010), and pleotrophic gene expression

This upregulated the levels of serum OPG and bone tissue TGF-1 protein, as well as p-Smad3/Smad3 in rats with osteoporosis, downregulated serum RANKL expression levels and elevated BMD, bone mineral salt content and femur biomechanical indices, including elasticity modulus, maximal load and yield load values in rats with osteoporosis

This leads to the accumulation of intracellular HO, triggering oxidative stress and ferroptosis in the cells

This led to the idea that niacin could be causing some other negative effect. In a study published in early 2024, Dr