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This helps maintain the safety and effectiveness of the medication for use

Effect of aging on the tendon structure and tendonassociated gene expression in mouse foot flexor tendon

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Here, we propose the misfolding of the mutant ATXN3 protein and the subsequent aggregation process, with eventual deposition of insoluble intracellular aggregates, to be the hub of the pathogenic process, and discuss how protein dyshomeostasis impacts key cellular pathways causing dysfunction and disease (Fig

Z.LiJ.CaiW.WuD.et al (2023)